sections include:
what causes Parkinson's disease?,
diagnosing Parkinson's disease,
treating Parkinson's disease
Parkinson's disease (PD) is a progressive
neurological disorder that most commonly develops
between the ages of 55 and 65. First described in
England in 1817 by James Parkinson, PD today afflicts
around 1 million individuals in the United States and
approximately 1 percent of those over 55; approximately
50,000 new cases are reported annually. The disorder’s
cardinal features are difficulty in initiating movement
(akinesia), slowness of movement (bradykinesia),
muscular stiffness (rigidity), and tremors at a rate of
four to five per second. As the disease progresses and
these symptoms become more pronounced, people who suffer
from it may have difficulty walking, talking, and
writing.
The onset of PD, which is typically subtle and
gradual, is most often unilateral, affecting only one
side of the body, with tremors the most common symptom.
In its earliest stages, doctors may miss the diagnosis
if a person does not have the characteristic resting
hand tremor. Some of the other common earliest symptoms
are decreased eye blinking, reduced facial expression,
and shrinking handwriting (micrographia). This early
period might last one to two years before more
pronounced symptoms appear. Tremor, slowness of
movement, and stiffness usually cause a person to seek
medical help.
Doctors describe the tremor as “pill rolling,”
meaning the motion one makes in rolling a pill across
the tips of the fingers with one’s thumb; it is present
at rest and is frequently a source of
embarrassment. The tremor typically disappears with
movement; driving is not affected early on. Other
difficulties are slowness, clumsiness, and increasing
fatigue in carrying out normal activities, as well as
stiffness, or increased muscle tone (rigidity). A person
with Parkinson’s disease typically develops a flexed
posture, bending forward at the waist, with the arm on
the affected side slightly flexed.
In addition to motor problems, people with PD
commonly suffer from depression and anxiety.
Depression affects as many as half of the patients.
It may occur at any stage of the illness but is often
missed because the decreased blinking and reduced facial
expressions that are characteristic of the disease can
mask depression. Or it may go untreated because of the
inappropriate expectation by both patients and
physicians that patients with Parkinson’s should be
depressed because of their condition.
Up to 30 percent of people with PD suffer some degree
of cognitive impai
or poor planning—usually with advanced illness, and
sometimes proceeding to dementia. But people with PD can
develop similar problems from other causes:
depression,
drug side effects and unrelated conditions, such as
Alzheimer’s disease. Doctors must therefore rule out
those other causes before assuming that a person’s
difficulties in thinking are related to PD.
Sleep disturbances are common in PD at any stage,
with many people getting fewer hours of sleep or
sleeping poorly. These individuals are often
significantly less alert during the day, compounding
their other challenges. Many factors may play a role in
these sleep disturbances, including side effects of
drugs, difficulty turning in bed, restless legs, vivid
dreaming, and sleep apnea (the repeated obstruction of
breathing). Correct diagnosis and treatment of sleep
disturbances can greatly improve daytime functioning.
Other symptoms of PD include sexual, urinary, and
bowel dysfunction. These, too, often respond to
appropriate drug therapy. Speech becomes low-pitched and
indistinct; the skin may develop seborrhea.
The course of PD varies considerably. Those who have
tremor as their major symptom may do exceedingly well
for a decade, while others may progress rapidly to more
disability over several years. Most people respond
excellently to drug treatment for the first three to
five years, but thereafter enjoy less benefit because of
drug-related complications, especially involuntary
movements (dyskinesias) and “wearing off” of drug
effects.
As the disease progresses, patients have increasingly
frequent and severe difficulties with gait and balance.
One hallmark of PD is the episodic “freezing” of gait: a
patient will be walking and will suddenly be unable to
take another step. Postural instability is one of the
most disabling and difficult-to-treat features of PD, as
well as a major factor contributing to falls, injuries,
and dependence.
PD is not generally fatal by itself, but people with
the disease do tend to have shorter life expectancies
than normal. Especially in later stages of the disease,
risks include choking, pneumonia, and severe and fatal
falls.
What Causes Parkinson's Disease?
PD is one of the best understood neurodegenerative
disorders. The pathological hallmarks of this disorder
include the degeneration of a small group of
dopamine-containing neurons in a part of the brain stem,
in a structure called the substantia nigra. Dopamine is
a chemical messenger that is released in portions of the
basal ganglia involved in the control of movement. Loss
of dopamine causes nerve cells to fire abnormally and
excessively, which disrupts the functioning of other
portions of the motor system that are directly
responsible for movement, causing excess neuronal
activity in some areas and diminished activity in
others. Because these disruptions are so specific to
movement, some scientists suggest thinking of PD as a
“circuit disorder.” Loss of dopamine neurons in the
substantia nigra occurs gradually over many years, and
PD symptoms develop only when 50 percent to 80 percent
of those cells have been lost and compensatory
mechanisms fail.
Many researchers are intensely studying the causes of
cell loss. For instance, “oxidative stress” caused by
cell-damaging free radicals, possibly through abnormal
breakdown of dopamine, is thought to be at least partly
responsible for the deaths of nigral cells. Excessive
accumulation of the protein alpha-synuclein, which is
often found clumped in dying nigral cells, is also
believed to play a role. Excess iron and other metals
are also often found in affected brain regions. The
synthetic opiate MPTP can cause Parkinson’s-like
symptoms, and, after being accidentally discovered in
drug users in the 1980s, became a chemical used to model
the disease in experimental animals. Other toxins and
pesticides have also been implicated in the origin of
PD.
Scientists are also studying the role of extranigral
changes in the brain, or those occurring outside the
substantia nigra, particularly inflammation and
neurodegeneration in the locus coeruleus, dorsal motor
nucleus of the vagus, and nucleus basalis.
It is generally believed that the vast majority of PD
cases result from an interaction between genetic and
environmental factors, but the relative roles of
heredity and environment are still uncertain. PD strikes
men and women in almost equal numbers across social,
economic, and geographic boundaries. Studies have shown
that the disease risk is higher among those who live in
rural areas, drink well water, or are exposed to
pesticides. Recent research has uncovered specific
genetic mutations in a small number of familial forms of
PD. For instance, mutations in the alpha-synuclein gene
can cause a rare inherited form, and chromosomal areas
known as PARK3, PARK8, PARK9, PARK10, and PARK11 have
also been linked to Parkinson’s susceptibility. While
this provides new insight into the disease’s causes, it
does not in itself explain the vast majority of
cases—approximately 95 percent—that appear
“spontaneously,” without obvious genetic triggers.
How the loss of dopamine in the brain leads to the
symptoms of slowness, stiffness, tremor, and the other
features of PD has received considerable study. This
research has led to new medical and surgical treatments
for the disease’s symptoms.
Diagnosing Parkinson's Disease
Parkinson’s disease is a clinical diagnosis; that is,
there are no specific medical tests for the illness.
Though generally not sensitive enough to directly
observe Parkinson’s-related brain changes, neuroimaging
with positron-emission tomography (PET) or
single-photon-emission computed tomography (SPECT) may
help rule out other conditions that clinically resemble
PD. These syndromes are often difficult to distinguish
from “atypical forms” of parkinsonism or “parkinsonism
plus.” These conditions share some of the four
primary symptoms described at the beginning of this
section, but not all are the result of losing
dopamine-producing brain cells. It is important to
distinguish PD from other types of parkinsonism because
treatment and prognosis differ significantly. Here are
some indications that a person’s symptoms are not
due to PD:
- The presence of clinical findings indicating
other disorders.
- Gait and balance difficulties appear early
rather than later.
- The symptoms do not respond satisfactorily to
antiparkinson medications (levodopa or dopamine
agonists).
- Imaging studies (magnetic resonance imaging,
PET) show that structures other than the basal
ganglia are involved in a person’s difficulties.
Scientists are now experimenting with radiotracers
and PET to directly track changes in the nigral areas of
the brain; many Parkinson’s patients show abnormalities
on such scans. Recently, tensor diffusion imaging of the
substantia nigra also showed promise. Some experts
estimate that diagnosis levels remain very low, with as
many as 20 undiagnosed people with Parkinson’s for every
known case.
Treating Parkinson's Disease
Although there is no cure for Parkinson’s, people can
be treated effectively for many years with medication,
physical exercise, and conditioning. Surgical approaches
provide new hope for patients for whom drugs no longer
control symptoms adequately. The goal of these
treatments is to maintain active functioning and quality
of life.
Advancing the medical treatment of PD is one of the
major accomplishments of modern medicine. Until the
mid-sixties, the drug treatment of PD consisted mainly
of anticholinergic drugs. These medications are still
occasionally used to treat tremors, but they cause
considerable side effects: memory impairment, visual
blurring, and urinary difficulties. The discovery that
people with PD have less dopamine in their brains led to
the use of the dopamine precursor, levodopa. On entering
the brain, this substance is chemically transformed into
dopamine, which acts on the dopamine receptors in the
basal ganglia. Levodopa therapy has become a mainstay of
treatment. It vastly improves the quality of life of
people with PD and has led to a nearly normal life span.
Soon after its introduction, however, it became
evident that levodopa does not arrest progression of the
disease. Even more problematic, it can lead to
drug-induced motor impairment (dyskinesias) and
cognitive complications. The introduction of alternative
drugs has helped lessen and delay these complications.
Levodopa is now given together with a drug called
carbidopa, which prevents the metabolism of levodopa in
the body. This reduces the side effects of nausea and
vomiting and maximizes the amount of levodopa that
enters the brain. Newer drugs that directly activate the
brain’s dopamine receptors (called dopamine agonists)
are now prescribed before levodopa because they appear
to delay and mitigate drug-related complications.
Amantadine is quite effective at reducing the severity
of drug-induced dyskinesias.
Attempts are under way to find so-called
neuroprotective treatments that will delay or arrest the
progression of PD. Approaches that use substances known
as growth factors and antioxidants are in this group.
Studies using experimental models of PD have shown that
destroying the part of the basal ganglia called the
subthalamic nucleus can immediately reduce stiffness and
tremor and improve mobility in the limbs on the opposite
side of the body. This discovery renewed interest in
surgical approaches to treating PD. To control tremor
and rigidity, doctors can also perform a thalamotomy, in
which a small section of the thalamus, a part of the
brain that relays signals coordinating movement, is
destroyed.
Another surgical procedure, pallidotomy, targets the
globus pallidus, a part of the basal ganglia whose
output leads directly to the symptoms of PD. All these
surgeries carry some risk of damage to adjacent brain
structures, but recent studies have found that they can
be highly effective in alleviating symptoms and reducing
side effects. It is as yet unknown how long the
beneficial effects will persist.
Another procedure, which mimics the effect of lesions
without actually destroying brain tissue, is deep brain
stimulation (DBS). DBS, which has been approved by the
Food & Drug Administration for treating PD, uses a
device similar to a cardiac pacemaker. The surgeon, with
imaging and physiological guidance, inserts an electrode
into the subthalamic nucleus and the globus pallidus
internal (GPi) that is connected to a small pulse
generator placed under the skin. The pulse generator can
be adjusted so that the optimal benefit is achieved by
varying the voltage, frequency, and pulse duration, a
process that usually takes several months. The
advantages of this method over surgery include the
ability to adjust the generator or stop using it. DBS is
generally employed only for those who do not or no
longer respond to medication or who suffer from severe
side effects. Studies have found that DBS is highly
effective for treating PD, working better than standard
drug therapy, but carries a higher risk of side effects,
mostly of complications from the brain surgery. Interest
in the treatment remains high, though, and more than
40,000 people have now undergone DBS surgery to treat
tremor or PD.
People with PD should consider having surgery before
the disease takes too great a toll on their
independence, employment, and self confidence. Younger
people with unilateral symptoms and intractable
drug-induced dyskinesias are the best candidates for
surgery, but there is no true age limit or lack of
substantial benefit for older individuals. Patients who
have developed dementia or have atypical parkinsonism
are not suitable candidates. Before they consider
surgery, or if they experience complications related to
treatment, patients should consult a movement disorders
specialist. All surgeries carry risk of severe side
effects, including infection and bleeding in the brain,
but brain surgeries also can cause neuropsychiatric side
effects including depression, personality changes, and
cognitive dysfunction.
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News on Parkinson's
Pesticides Linked to Parkinson’s
(June 2008) -- Another large study shows that people
with Parkinson’s disease are significantly more likely
to report a history of pesticide exposure than are
people—including other family members—who do not have
the disease.
Facing Up to the ‘New Face’ of Parkinson’s
Disease (May 2008) -- An interview with
Anthony E. Lang, M.D., who has proposed the concept of
Parkinson’s diseases (plural).
Parkinson's Research Moves Beyond Dopamine
(March 2008) -- After nearly 50 years of focusing on
dopamine, the brain transmitter system that fails in
Parkinson’s disease, experts in the field are
increasingly convinced of the need to consider other
possible culprits to solve the riddle of this disabling
neurodegenerative disorder.
TRANSPLANTATION
Another surgical approach to treating PD is to
replace the missing dopamine neurons with new cells
derived from fetal or other tissue. This approach
remains experimental. One of the more promising recent
avenues of research for Parkinson’s disease, and for
other neurodegenerative disorders, involves what are
called stem cells. These cells can develop into the
different sorts of cells that make up our bodies. Stem
cells can be either embryonic or adult. Embryonic stem
cells are derived from the earliest stage of development
and have the greatest potential because they can lead to
any tissue or cell type in the body. Adult stem cells
are found in many tissues of the body—for example,
blood, skin, and even brain. These cells can generate
all of the cell types of their respective tissues. Blood
stem cells, for instance, can become red cells, white
cells, or platelets. Adult stem cells appear to have
some capacity to cross tissue lines, but less than that
of embryonic stem cells.
Stem cells hold potential for medicine in several
ways. First, by observing and testing how they behave,
we can learn more about how we normally develop and how
cellular growth can go wrong, as in cancer. We might
also be able to use these cells to create tissues on
which we can test new drugs without endangering human
(or animal) lives. But most of the public excitement
over stem cell research involves the potential of
growing cells to replace those the body lacks.
Researchers have begun to manipulate human stem cells to
produce new dopamine-producing neurons; people with PD,
who are missing those crucial cells, could have a new
supply inserted into their brains.
There are many challenges to overcome before we
achieve such therapies, however. One difficulty is
learning exactly how stem cells are guided to become one
type of cell or another. Another is figuring out how to
graft the new cells in place so that the body does not
reject them and so that they work as the brain and body
need them to.
HEALTH STRATEGIES WITH PARKINSON’S DISEASE
Treating Parkinson’s disease is not exclusively the
doctor’s job; the patient can do a lot to stay as well
as possible for as long as possible. Here are three
things patients can do for themselves.
■ Exercise: For people with Parkinson’s, regular
exercise and physical therapy are essential for
maintaining and improving mobility, flexibility,
balance, and a range of motion and for warding off many
of the secondary symptoms that can occur with the
disease. Exercise is as important as medication for the
management of PD.
■ Support groups: These groups play an important role
in the emotional well-being of patients and families.
They provide a caring environment for asking questions
about Parkinson’s, for laughing and crying and sharing
stories and getting advice from other sufferers, and for
forging friendships with people who understand each
other’s problems.
■ Staying active: PD seems to advance more slowly in
people who remain involved in their pre-Parkinson’s
activities, or who find new activities to amuse them and
engage their interests. In a word, getting joy out of
life has proved to be good for health.
Adapted from
www.pdf.org (Parkinson’s
Disease Foundation)
PARKINSON'DDS D
DISEASE AND DIET
Diet in Early Stages of PD
Tips on What to Eat
From
Patrick McNamara, Ph.D., former About.com Guide
About.com Health's Disease and Condition content is reviewed
In the early stages of
Parkinson's disease (PD), no special dietary changes are
typically required. You may want however to increase your intake of
foods rich in antioxidants.
Antioxidants are those chemicals that scavenge and eat-up
so-called ‘free radicals’ –- tiny molecules that circulate in your
tissues and damage those tissues. Free radicals have a special
affinity for cells that produce
dopamine. So the greater the number of antioxidants in your
system, the fewer the number of circulating free radicals.
Theoretically that should reduce the rate of loss of dopamine cells
over time.
So what foods contain a lot of antioxidants? Fruits and
vegetables -- especially the darkly colored fruits and vegetables.
Some examples include leafy green vegetables (such as spinach),
broccoli, tomatoes, carrots, garlic, red kidney beans, pinto beans,
blueberries, cranberries, strawberries, plums and apples. Tea --
especially green tea and black tea -- contains a lot of
antioxidants. Red wine contains antioxidants. Dark juices like
pomegranate and blueberry juices are rich in antioxidants.
Omega-3 fatty acids are an essential nutrient for most tissues in
your body so you want to make sure to consume adequate amounts of
these nutrients. Fatty fish like mackerel, lake trout, herring,
sardines, albacore tuna and salmon are high in two kinds of omega-3
fatty acids, eicosapentaenoic acid (EPA) and docosahexaenoic acid
(DHA). It is probably a good idea to add fish to your diet if you
are not already eating fish on a regular basis.
You also need to get adequate sources of calcium, magnesium, and
vitamins D and K to strengthen skin and bone. You can usually obtain
these nutrients from dairy products like yogurt and milk. You need
some amount of sunlight to get enough vitamin D.
Here are some ways to include these healthy foods in your
diet:
- Blueberries, raspberries and blackberries can be stirred
into vanilla yogurt for a delicious dessert. Or they can be
blended with fat-free yogurt and ice in a blender to make a
smoothie. Fruit smoothies can also help prevent the constipation
associated with some PD medications.
- I never liked spinach that much until I found a way to
combine it with foods I do like, such as salad or rice or
scrambled eggs. Happily I like these combinations better than
any of the stand alone alternatives. I stir chopped, fresh
spinach, tossed in olive oil into my salads or into steamed
brown rice. I also sometimes add raisins to that rice and
spinach dish.
- Carrots are loaded with a potent antioxidant called
beta-carotene. Cooked, steamed or pureed carrots liberate the
antioxidants or somehow make them easier to absorb. Cooked
carrots are often more tasty as well. I like my carrots steamed
and then slowly cooked in the juices of whatever meat dish I am
having.
- Vitamin E is a potent antioxidant and is found in some nuts
in whole grains. Although studies on the anti-PD effects of
vitamin E have yielded only discouraging or mixed results
vitamin E should nevertheless be a part of your diet. I get most
of my vitamin E from whole grains. I cook and steam some whole
grain like brown rice, cous cous or bulgur wheat. Then I add
items like raisins or cranberries, chopped parsley or spinach,
and olive oil.
Sources:
Weiner, W. J., Shulman, L.M. and Lang, A. E. (2007). Parkinsons
Disease, Second Edition, A Complete guide for patients and families.
Johns Hopkins Press Book, Baltimore.
Marczewska A, De Notaris R, Sieri S, Barichella M, Fusconi E,
Pezzoli G. Protein intake in Parkinsonian patients using the EPIC
food frequency questionnaire. Movement Disorder. 2006
Aug;21(8):1229-31.
Parkinson's Disease Alternative Medicine
Hope is a powerful thing. Without hope for a better future, the will
to live is lost and recovery, no matter what the affliction, is
impossible.
Imagine then, being told by your doctor that you have a progressive
and degenerative disease that robs you of the ability to control your
own body. A disease for which there is no cure.
This is the reality for those diagnosed with Parkinson's disease.
Hope is offered in the form of medication such as Levodopa. Levodopa
acts to restore levels of dopamine in the brain. The lack of dopamine is
the primary reason for the symptoms associated with the condition. Since
it first began being administered in the 1960's, it has lessened much of
the suffering experienced by millions of people throughout the world,
and is recognised as the "gold standard" in medical treatment of the
disease. However, it can not completely reverse the symptoms, and like
all drugs, is more effective in some than others.
Consequently many have sought hope in alternative treatments.
Parkinson's disease has been a recognised ailment in virtually all
cultures since ancient times. Many of these ancient treatments are
becoming popular in the west and are increasingly validated by western
medicine.
Ayurvedic medicine - This has been practiced in India for 5000
years. Parkinson's symptoms are mentioned in ancient text under the name
Kampavata. Ayurvedic medicine is a comprehensive system placing equal
emphasis on diet, exercise, meditation, massage and herbs. One such
herb, Mucuna Puriens, is gaining attention in conventional circles as
its effects mimic synthetic Levodopa, with fewer side effects.
Broad beans- Australian researchers discovered that broad
beans are also an extremely effective natural source of L-dopa. The
highest concentration of L-dopa is found in the pod so they are most
effective when consumed whole.
St John's Wort- Dopamine influences positive feelings in the
brain, and since dopamine levels are low in Parkinson's patients,
depression is often a symptom. St John's Wort is a herb that has been
used in Europe for many years. It has been proven to be effective in
alleviating depression and insomnia.
Botulinum toxin A- This is a bacterium that causes food
poisoning (botulism) but has proven to be effective in reducing hand,
head and voice tremors when in a weak solution.
Coenzyme Q10 (CoQ10) - This has been shown to have an effect
on the symptoms of Parkinson's Disease, however it is unclear whether it
actually slows the disease or simply temporarily alleviates symptoms.
The drawback is the massive dose required. The effective dose is
approximately 1,200 milligrams a day, well above the 60 to 90 milligrams
recommended by many alternative therapy advocates.
Accupuncture- Used for centuries in China to correct energy
disturbances in the body. It has become a popular method of treatment
for Parkinson's sufferers the world over. So far there are no placebo
controlled studies that show acupuncture can treat the motor control
symptoms of the disease, but there is some evidence that it can assist
with sleep disturbances. There is much anecdotal evidence to suggest
that it may be effective in increasing feelings of well being and
relaxation.
Massage- While not treating the symptoms directly, it can help
reduce some of the discomfort associated with muscle stiffness that is
commonly experienced by patients.
Alternative treatments for many diseases come and go. Some become
fashionable for a short while only to be discredited and discarded.
Others accumulate a growing body of scientific and popular support. It
is to a large extent simply a matter of trial and error. Most
alternative treatments are harmless, but some herbal remedies may
interfere with medication, so be sure to consult your health
practitioner for advice.
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The content of this web site is intended to convey
general educational information and should not be relied upon as a
substitute for professional healthcare advice.
More information.
2002-2009©Parkinson's Disease Information.
DO NOT TAKE
STATIN DRUGS
STATIN DRUGS CAUSES WEAKENING OF THE HEART
AND CAUSES PARKINSON'S DISEASE
DO TAKE THIS
CO-Q 10
START WITH 100 MGs OF co-q 10
it isi hard on the stomach so increse the dosage
slowly
increase 100 mg per week until you get to 400
then increase it only once a month until your stomach
gets used to the grease in it. The stomach
doesn't tolerate
the grease in the pill very well - thus you must
increase
only once a month after 1200 mgs, then ncrease only
once
every 4 months or so.
3,000 mg of co-q 10 will cure Parkinnson's disease as
well as
give you heart the tone of muscle it needs.
most people do not have enough co-q 10 naturally in
their bodies.
this information was given by Dr. Jimmy who has
studied these
problems for many years.
LAYING ON OF HANDS
There is more than one way to do this process.
It can be done with religious prayer, or with simple words, or with
formal programs, such asa REIKI HEALING or other methods similar to
this.
The laying on of hands is a religious ritual
that accompanies certain
religious practices, which are found throughout the world in varying
forms.
In
Christian churches, this practice is used as both a symbolic and
formal method of invoking the
Holy Spirit primarily during
baptisms
and confirmations,
healing services,
blessings, and
ordination of
priests,
ministers,
elders,
deacons, and other church officers, along with a variety of other
church
sacraments and holy ceremonies
SEE
http://en.wikipedia.org/wiki/Laying_on_of_hands
"Laying on of hands" is a biblical action; however, there is no biblical
mandate requiring the physical laying on of hands for a particular
spiritual ministry. Jesus certainly laid His hands on many of those He
healed; however, He also healed without laying His hands on people. In
fact, there were times when He was nowhere in the vicinity of those He
healed.
Matthew 8:8 describes Jesus healing the servant of the centurion
without going near the centurion’s house.
Here are two instances to consider: in one case the Holy Spirit bestows
the gift of speaking in tongues with the act of an apostle's laying on
of hands, and in the other case He does so without the laying on of
hands, but simply through the apostle's preaching.
"Paul said, ‘John's baptism was a baptism of repentance. He told the
people to believe in the one coming after him, that is, in Jesus.’ On
hearing this, they were baptized into the name of the Lord Jesus. When
Paul placed his hands on them, the Holy Spirit came on them, and they
spoke in tongues and prophesied" (Acts
19:4-6).
"While Peter was still speaking these words, the Holy Spirit came on all
who heard the message. The circumcised believers who had come with Peter
were astonished that the gift of the Holy Spirit had been poured out
even on the Gentiles. For they heard them speaking in tongues and
praising God" (Acts
10:44-46).
First Timothy 5:22 says, "Do not be hasty in the laying on of hands,
and do not share in the sins of others. Keep yourself pure." The thought
here is not so much in cautioning the physical action of laying on of
hands but to urge care in bestowing the responsibility of spiritual
leadership (however it it is done). It is not to be done "suddenly" or
without due consideration.
Undoubtedly, the laying on of hands in the early church was a means of
connecting the message with the messenger, or the spiritual gift with
the gifted giver. It provided a "sign" authenticating him through whom
the physical manifestation of a spiritual gift was bestowed. We need to
understand very carefully that there are no magical biblical formulas
for the ministry of the church. Laying on of hands has no power in
itself. Laying on of hands is only
used by God when it is done in agreement with God's
Word.
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NOTE FROM DEE I have studied Reiki
myself twice at great expense and have two Master certificates to
prove it. Reiki is an old ancient Japanese method of
healing,and the lessons are very expensive. They say that
Reiki can only be activated by a Master teacher, I personally don't
agree as the healing itself comes from the God source, not from
onenself. The God source does not withhold healing because you
haven't paid thousands of dollars for it.
Reiki is very similar to the laying on of hands except that one uses
specific positions on the body, nt just on the head or shoulder as
in the above picture, but Reiki energy goes where it needs to go and
need not be directed. Reiki can also be done at a distance,
just like prayer can be done at a distance. The method is
similar.
I will just give links to the best places to get
training.
THIS IS STEVE MURRAY"
http://www.google.com/#sclient=psy-ab&hl=en&source=hp&q=reike+steve+murray+&pbx=1&oq=reike+steve+murray+&aq=f&aqi=g-l1g-lv3&aql=&gs_sm=e&gs_upl=13917l25497l0l26195l19l19l0l0l0l0l685l7464l2-4.9.3.3l19l0&bav=on.2,or.r_gc.r_pw.,cf.osb&fp=8bdce738706c865e&biw=607&bih=236
REIKI HAND POSITIONS VIDEOS
http://www.youtube.com/watch?v=Xw9EleHuXsI
DR. ERIC PEARL - HANDS ON HEALING
http://search.aol.com/aol/search?s_it=topsearchbox.search&v_t=comsearch50&q=DR+ERIC+PEARL
DR. ERIC PEARL VIDEOS
http://www.youtube.com/results?search_query=DR+ERIC+PEARL&aq=o
USUI REIKI
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